Multiple Sclerosis (MS) is a devastating neurological disease. MS patients often experience neurologic dysfunction believed to be the result of infiltration of immune cells, inflammation of the central nervous system and eventual nerve damage.
Researchers at the University of California, Irvine have identified a monoclonal antibody-based method that decreases immune cell infiltration into the CNS and improves neurological function in a mouse model of MS.
This antibody may offer an attractive method for reducing CNS inflammation in humans and treating inflammatory demyelinating diseases such as MS.